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Brown Sauce and honest reporting

The British are fond of a condiment called Brown Sauce. The product itself leaves me unmoved, but the thing I find interesting about Brown Sauce is that it purports nothing about itself whatsoever, other than a description of its colour. It claims no link to any known product of nature, just a factual statement about its appearance. Consider, for instance, tomato ketchup. If an independent lab discovers that a ketchup is, in fact, only 5% tomato and 95% starch, sugar, salt, and flavor enhancer 621, people will be justifiably irate about the “tomato” claim. If, on the other hand, Brown Sauce is eventually proven to be made from asbestos and drowned kittens, the manufacturers can quite rightly state that they only said it was brown.

The same kind of plain speech is often missing in surgery. The truth has often been a casualty in the patient consent process due to a combination of ignorance, fear, avarice, or ego on the part of the surgeon. Whatever the motivation, when we explain rates of risks and benefits to the patient before us, many of us are not giving an honest report of our own outcomes. In the case of the battle between robotic and open radical prostatectomy, for example, real-world complication rates are often ignored in favour of Walsh’s rates on one side, and Patel’s on the other. Surgeons are certainly not all the same. If you have ever considered who you would allow to perform surgery on yourself the chances are you have written a very short short-list. When we tell a patient that the rate of complication x from procedure y is only 5% and we have not audited our own outcomes, we are likely giving the rates produced by the high-volume specialist centres that had the expertise, numbers, and clout to get their rates published in a reputable journal. Most surgeons do not work in those centres.

There is an on-going debate on whether hospitals should be compelled to publish their procedure-specific outcome data, so that the public can make informed decisions about their surgical care. I think this misses the point. Yes, there are potential hazards to compulsory publishing; centres of excellence may have worse outcomes than others due to operating on the sickest patients with the slimmest hopes of success, one major complication in a lower volume centre can skew the data, and there is the potential to develop a culture of suspicion and dishonesty, but the real point is more personal. We should honestly report to the patient in front of us from our own results as a matter of honesty and ethics, regardless of hospital policies. We can then (hopefully) reassure them that our outcomes are comparable to those published, and they can expect good quality care from us. If we cannot reassure them of this, our audit process will inform us of our shortcomings and we can seek to address them. We might even consider leaving certain procedures to a colleague who is better at it than us. A bitter pill, maybe, but arrogance is the enemy of improvement.

It can be a nuisance to collect and collate operative data. It can be painful to discover that we are not as good at something as we had assumed. Thankfully surgeons are mature adults who can take these challenges on the chin, and use the results to make our patient care better. Can’t we?

Otherwise, the information we give our patients is “pork-pies”, which is Cockney rhyming slang for lies, and no amount of Brown Sauce can make those pies palatable.

James Duthie is a Urological Surgeon/Robotic Surgeon. Interested in Human Factors Engineering, training & error, and making people better through electronic means. Melbourne, Australia @Jamesduthie1

 

A word of advice

I saw a patient recently who presented with a number of different symptoms on the background of a complex past medical history. I rang my senior who quoted me these words,

“Sometimes the questions are complicated but the answers are simple”

I’ll tell you where he got that quote from later

One CT scan later we had our diagnosis but it got me thinking about the advice we receive from senior colleagues.

 


During our formal urology training we are mentored by urologists who at times impart advice to enable us to become better. The words spoken in a timely manner or often a repeated manner can become etched into a young surgeon’s mind. 

Now for 2 stories:

Early in my surgical training I sat dejected in the professor’s office. A life threatening complication had occurred in one of our team’s patients after major open surgery. I was ready to throw the towel in, to no longer perform large operations to therefore avoid complications. I would be safe if I only ever performed minor procedures for the rest of my career.

“There are no such things as small surgeries only small surgeons”.

Spoken by one who had been there and done that and come out the other side. Years later near the end of my training I was lucky to operate again with this surgeon and thanked him for his words of advice. However as often happens in a moment of stress, similar I imagine to the moment a diehard fan meets their rock star idol, I proudly repeated back his words only for him to correct me again, as I had spoken them in the wrong order.

Story two took place after a Saturday ward round over coffee. Real coffee – (flat whites as we call them in New Zealand) in the hospital café where there was time to talk – no theatre list, no private clinic to rush off to.

“The cheapest mistake you will ever make is the complications of others”

I was implored to chase after the complications that happen to all patients in the hospital. So as to learn as much from them and how they were managed to avoid it in my own practice in the future. It challenged me to investigate, to read, to apply knowledge, to sit up in the monthly audit and to ask questions of why things happen. 

3 statements have helped guide my surgical training. Said in isolation they mean little and could even sound cliché. However coming from a respected mentor at an appropriate time in a personalised fashion they have proved of immense value. And the further beauty is they will have repeated value as I attempt to pass them on in the future. Which brings me back to the opening statement and the whereabouts of its origins. Indeed those words were not my consultants own but rather that of another doctor. Dr. Seuss to be exact and it only goes to show that advice is often where you least expect it.

(On further research I wonder if this is where he actually got the statement from

Advice’s You Need to Know About Hair Loss, According to Dermatologists

hair loss
SAKSIT SRISUKSAI / EYEEMGETTY IMAGES

If you’ve noticed patchy or thinning spots on your scalp or a surplus of hair strands on your hairbrush or in the shower, you’re not alone. More than half of all women will experience noticeable hair loss, according to the Cleveland Clinic. In order to put a stop to shedding, though, you have to figure out the root of the problem. “Hair loss is not a diagnosis,” says Yolanda Lenzy, M.D., M.P.H.,board-certified dermatologist and licensed cosmetologist in Chicopee, Massachusetts. “Hair loss is a symptom. Once you get a specific diagnosis, then you can know the causes associated with that diagnosis.” You can check some of the best dermatologist melbourne on doctor to you site.

For starters, know that the term “alopecia” refers to all kinds of hair loss. There are two main types of alopecia, and then a variety of forms of hair loss within those two categories. In cases of non-scarring or temporary hair loss, the missing hair will eventually grow back. With scarring or permanent hair loss, permanent damage is done to the hair follicles so they won’t grow back. “With scarring hair loss, the goal is not for it to grow back, but to stop the progression,” says Dr. Lenzy. Below, are seven different kinds of alopecia —knowing which one you’re suffering from will help determine the proper treatment.

woman combing hair
GETTY IMAGES

Non-scarring (Reversible) Forms of Hair Loss

Androgenetic alopecia

When people talk about male or female pattern hair loss — typically a receding hair line in men or thinning at the crown in women — that’s androgenetic alopecia. It’s the most common form of hair loss among all people. In fact, research shows that more than 50% of women will develop androgenetic alopecia by the age of 80. “It can come from either side of the family in men or women, skip a generation, and start earlier in the next generation that the one before it,” says dermatologist Carolyn Goh, M.D., Health Sciences Assistant Clinical Professor at the David Geffen School of Medicine and Director of the Hair and Scalp Disorder Clinic at UCLA. “However, some people have pattern hair loss without a family history of it.” While some women start showing signs of androgenetic alopecia in their teenage years, others won’t experience it until their 50s or 60s. “When nearing menopause, the decrease of estrogen means you have unopposed testosterone,” says Dr. Lenzy. “That elevated testosterone can convert to a hormone called dihydrotestosterone (DHT), which contributes to the thinning of the hair follicles — the follicles actually get smaller in this particular form of hair loss.”. If you have straight hair then consister a permed texture you should buy kinky straight hair.

MORE FROM GOOD HOUSEKEEPING

Telogen effluvium

Telogen effluvium is just a fancy name for excessive hair shedding — an annoyance that many people will experience at some point in their life. “A common cause is stress, usually meaning major life stressors or physical stressors like surgery, medication (including over the counter ones and supplements), weight loss, or a death in the family, to name a few,” says Dr. Goh. “It usually starts three to six months after a stressor and then lasts for three to six months.” Hypothyroidism and iron deficiency can also trigger telogen effluvium. “The beautiful thing about it is 70% of your hair strands are still in the anagen or growing phase,” adds Dr. Lenzy. “Because the hair follicles work in a cycle, you won’t go bald.”

Alopecia areata

This type of hair loss affects about 2% of people and usually appears as round smooth circles anywhere on the head without any redness, itching, or pain. “Alopecia areata is thought to be caused by an autoimmune process,” says Dr. Lenzy. “The body’s immune system makes some mistakes and produces T cells that attack hair follicles.”

Traction alopecia

Thinning and bald patches at the temples or where hair is frequently pulled tight can indicate traction alopecia. “This very common form of hair loss is caused by haircare and hairstyle practices — practices which place excessive tension or weight on the follicles like braids, ponytails, hair extensions, or locs.”

Scarring (Permanent) Forms of Hair Loss

Central centrifugal cicatricial alopecia (CCCA)

“Central centrifugal cicatricial alopecia tends to start on the top of the head with breakage and thinning, and often with some tenderness of the scalp,” says Dr. Goh. “It gradually spreads outward and can cause permanent hair loss.” CCCA is especially common among Black women. “Some recent studies have found that about 25% of people with this form of hair loss have a genetic mutation in one of the proteins that’s responsible for the formation of the hair follicle,” says Dr. Lenzy. On top of that, she notes that the same haircare practices that create tension and cause traction alopecia also contribute to CCCA.

A Rather Nasty Surprise

Recently, I encountered, and indeed I actually caused, a complication of robot-assisted radical prostatectomy (RARP) which was new to me, and one which I felt that I should share with other surgeons.

PM, a 60-year old teacher, underwent a completely routine RARP, which took less than 2 hours to perform on a Saturday morning. During Sunday night he developed severe abdominal pain and distension. By Monday morning he was in distress with rebound tenderness and marked tachycardia. A CT scan was requested, which revealed a caecal volvulus. A laparotomy by a general surgeon confirmed the diagnosis and an urgent right hemicolectomy was undertaken. The patient made an uneventful recovery and, I am pleased to say, is still speaking to me. Histology confirmed an ischaemic caecum twisted on its rather thickened mesentery, with no perforation present. The prostate itself contained a Gleason 3+4=7 adenocarcinoma, without evidence of extra-prostatic extension.

Although robotic assistance provides the benefits of very precise, virtually bloodless surgery, with 10 times magnification and 3D vision, it also carries the risk of a specific set of complications. These need to be dealt with promptly and efficiently and can usually be completely resolved. Failure to recognise post-operative problems, such as bowel injury, intra-abdominal bleeding or port-site hernia, however, can place the patient in severe and increasing jeopardy. We recently published an article in the BJUI entitled “Lessons Learned from 1000 robot-assisted radical prostatectomy” in which we discussed how many of the problems could be avoided, and, if they occur how they can be best dealt with. One key message is the importance of an early CT scan to diagnose the nature of a post-operative problem, rather than crossing fingers and hoping things will settle.

I am hoping that this blog, and the BJUI article mentioned above, will stimulate other surgeons to discuss openly and frankly the problems that they themselves have encountered, either with regular laparoscopy or with the da Vinci robot, and how they dealt with them. Learning the lessons, not only from one’s own errors and omissions, but also from those of others, seems the best way to become, and continue to be, a safe and successful surgeon.  

 

Roger Kirby, The Prostate Centre, London

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